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We next investigated whether altered insulin production in Tmprss2 / mice led to enhanced local insulin signaling in the intestine

Our present observations are in keeping with our previous findings (17,30,31) indicating that the production of free radical, enhanced lipid peroxidation and an impairment of antioxidizing enzyme activity, and the increased generation of cytokines, such as IL-1 and TNFalpha, contribute to the pathomechanisms of stress-induced gastric lesions